For Patients & Caregivers
How It Works
Cellfood has not been shown to treat or cure cancer.
Cellfood is a line of dietary supplements sold over the internet and in health food stores. Promoters claim that it contains minerals, enzymes, amino acids, and dissolved oxygen, which hyperoxygenates the body. Lab studies show that Cellfood has antioxidant effects, and causes some cancer cells to die. But these effects have not been shown in humans.
Supplements with antioxidant properties are not recommended during chemotherapy and radiotherapy.
For Healthcare Professionals
Cellfood is a line of dietary supplements and topical preparation manufactured by NuScience Corporation. The original formula contains colloidal minerals, amino acids, enzymes and dissolved oxygen (1). It is marketed as a free radical scavenger to help detoxify the body and to enhance athletic performance. Cellfood is also promoted to cancer patients based on the theory that cancer and degenerative diseases are caused by oxygen deficiency at the cellular level.
In vitro studies show that Cellfood has antioxidant (2) (3) and antiproliferative effects (4) (5). Small studies indicate that it may alleviate symptoms of fibromyalgia (6); when combined with chelation treatment, cellfood improves oxidative status and homocysteine metabolism in patients with neurodegenerative diseases (ND) (7), as well as in patients affected by ND showing aluminium intoxication (8).
Cellfood has not been studied in cancer patients. Supplements with antioxidant properties are not recommended during chemotherapy and radiotherapy.
Mechanism of Action
Cellfood demonstrated antioxidant effects in vitro (2) (3) and proapoptotic effects in human mesothelioma and colon cancer cells, which correlated with increased expression of tumor suppressor genes p53, p21, and p27; downregulation of the oncogenes c-myc and Bcl-2; and inhibition of activation of antiapoptotic protein kinase, Akt (4). Cellfood also showed antiproliferative effects in leukemia cells by inducing apoptosis and by altering the metabolism of cancerous cells through regulating the hypoxia inducible factor 1 alpha (HIF-1α) and glucose transporter 1 (GLUT-1) (5).